Safety of lornoxicam in G-6-PDH deficiency

[Safety of lornoxicam in G-6-PDH deficiency].

OBJECTIVE To assess the safety of lornoxicam in subjects with G-6-PDH deficiency. METHODS Open controlled 2-week in vivo study on lornoxicam 8 mg bid in subjects with G-6-PDH deficiency suffering from rheumatic diseases. RESULTS In 8 male patients with the Mediterranean form of G-6-PDH deficiency (mean age +/- SD, 54.3 years +/- 7.2) lornoxicam showed no influence on red blood cells (RBC) s...

Glucose-6-phosphate dehydrogenase (G-6-PD) deficiency.

G 6 PD Deficiency in Two Autochthonous Croatian Families

In Europe G6PD deficiency is encountered most frequently in Sardinia and Greece but may also be found in other Mediterranean countries (Sansone, Piga, and Segni, 1958; Szeinberg, Asher, and Sheba, 1958), including Yugoslavia (Fraser, Grunwald, and Stamatoyannopoulos, 1966). The occurrence of haemolytic anaemia after ingestion of fava beans in Yugoslavia was first reported as a clinical finding ...

Ascorbic-acid-induced haemolysis in G-6-PD deficiency.

THOUGH THE ORIGINS of erythrocyte glucose-6-phosphate dehydrogenase (G-6-PD) deficiency are rooted in human antiquity, the presence of drug-induced hemolysis in individuals subsequently felt to have G-6-PD deficiency was first reported in 1926 and the biochemical lesion responsible for this phenomenon elucidated in 1956 (1). In the United States, hemolysis induced by infection or drugs is the m...

G 6 PD deficiency induced hemolysis in diabetic twins.

We report hemolysis due to glucose-6-phosphate dehydrogenase (G-6-PD) deficiency in two Egyptian monozygotic brothers, at the onset of diabetes type 1. Hemolysis occurred following the treatment of hyperglycemia and ketoacidosis (one twin). It was related to unknown G-6-PD deficiency. The fall in glucose availability after the treatment of hyper glycemia is proposed as a possible etiology for h...